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DSIP

Emideltide, DSIP nonapeptide, Delta sleep-inducing peptide

Quick Stats
Studies 458
Trials 82
Score 1
1984 pubmed

Fibrinopeptide A binds Gly-Pro-Arg-Pro.

Root-Bernstein. R S RS; Westall. F C FC

Key Findings

  • The tetrapeptide Gly‑Pro‑Arg‑Pro binds to fibrinopeptide A.
  • The binding constant is about 10⁓ M⁻¹, indicating moderate affinity.
  • Nineteen related peptide controls were tested to confirm specificity.

Practical Outcomes

  • For now, there’s no direct way to use this peptide for longevity or performance. It’s a basic science step that could eventually guide anti‑clot or anti‑inflammation strategies, but more research is needed before any real‑world protocol can be recommended.

Summary

Researchers found that a tiny four‑amino‑acid peptide (Gly‑Pro‑Arg‑Pro) sticks to a part of fibrinogen called fibrinopeptide A, with a moderate binding strength. This helps map where the peptide might block fibrin formation, but the study didn’t test any health effects or dosing.

Abstract

The tetrapeptide Gly-Pro-Arg-Pro inhibits fibrinogen aggregation, probably by binding to the same sites used during initiation of fibrin formation. The Gly-Pro-Arg-Pro binding sites have not yet been identified. However, their possible sequence and locations have been predicted on the basis of the amino acid pairing hypothesis. One of these predicted sites is on fibrinopeptide A. We report here that nuclear magnetic resonance studies indicate that Gly-Pro-Arg-Pro binds to fibrinopeptide A with a binding constant, K, of ca. 10(4) per mol. We also report results of 19 related peptide combinations used as controls.

Study Information

Provider

pubmed

Year

1984

DOI

10.1073/pnas.81.14.4339