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GHRP-2

Pralmorelin, Growth Hormone Releasing Peptide-2, KP-102

Quick Stats
Studies 230
Trials 1
Score 2
2009 pubmed

Exaggerated response of adrenocorticotropic hormone to growth hormone-releasing peptide-2 test in Cushing's disease. Case report.

Sakihara. Satoru S; Kageyama. Kazunori K; Matsumoto. Atsufumi A; Ikeda. Hidetoshi H; Tsushima. Yuko Y; Naraoka. Maki M; Terui. Ken K; Nigawara. Takeshi T; Suda. Toshihiro T

Key Findings

  • s disease patient, a 100 ”g dose of GHRP‑2 raised ACTH from 66 pg/mL to 2,490 pg/mL (≈38‑fold).",
  • ,

Practical Outcomes

  • For biohackers using GHRP‑2, be aware that the peptide can sharply increase ACTH and potentially cortisol, especially if there’s an underlying pituitary abnormality. While the study isn’t about healthy performance enhancement, it suggests monitoring stress‑hormone levels if you notice unusual side effects. The finding is mainly a diagnostic insight rather than a new protocol for longevity or performance.

Summary

The report describes a single patient with Cushing's disease whose ACTH levels shot up dramatically after a standard GHRP‑2 test. The tumor had the GHRP‑2 receptor, suggesting the peptide can directly boost ACTH in certain pituitary tumors. For most people this isn’t a therapeutic finding, but it shows that GHRP‑2 can strongly affect the stress‑hormone axis.

Abstract

A 47-year-old woman presented with a pituitary microadenoma manifesting as typical Cushing's syndrome. The diagnosis was Cushing's disease based on the endocrinological findings. Plasma adrenocorticotropic hormone (ACTH) levels were greatly increased from 66 pg/ml to 2490 pg/ml (about 38-fold) in response to the administration of 100 microg human growth hormone-releasing peptide (GHRP)-2. GHRP receptor type 1a messenger ribonucleic acid was detected in the tumor. Therefore, GHRP-2 may stimulate ACTH via the GHRP receptor type 1a in pituitary ACTH-producing tumor. The GHRP-2 test, currently clinically available in Japan, may be a useful diagnostic tool for Cushing's disease.

Study Information

Provider

pubmed

Year

2009

DOI

10.2176/nmc.49.365