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LL-37

Cathelicidin, hCAP-18, FALL-39, CAP-18

An antimicrobial peptide of the cathelicidin family that provides innate immune defense by killing pathogens and modulating inflammation and wound healing.

Quick Stats
Studies 2230
Trials 95
Formula C205H340N60O53
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Utility 2
pubmed Oct 10, 2018

Synergistic microbicidal effect of cationic antimicrobial peptides and teicoplanin against planktonic and biofilm-encased Staphylococcus aureus.

Koppen. Bruce C BC; Mulder. Patrick P G PPG; de Boer. Leonie L; Riool. Martijn M; Drijfhout. Jan W J...

Researchers found that the natural antimicrobial peptide LL‑37 (and lab‑made versions inspired by it) works much better at killing Staphylococcus aureus, especially when paired with the antibiotic teicoplanin. This combo also breaks down tough bacterial biofilms, but it didn’t help against Staph epidermidis.

Utility 2
pubmed Nov 1, 2018

[Evaluation value of human antibacterial peptide LL-37 on the prognosis of elderly patients with sepsis].

Guo. Weina W; Wang. Chunmei C; Huo. Fangjie F; Li. Hongqiang H; Yan. Yanli Y; Xu. Shumin S; Xu. Huih...

The study measured the natural antibacterial peptide LL‑37 in older people with sepsis and found that it spikes early in the illness but falls as the condition gets worse. Lower LL‑37 levels around day 7 were linked to higher severity scores and a greater chance of dying within 28 days.

Utility 2
pubmed Dec 16, 2017

Prion Protein PRNP: A New Player in Innate Immunity? The Aβ Connection.

Lathe. Richard R; Darlix. Jean-Luc JL

The study shows that the normal prion protein (PRNP) may act like an antimicrobial peptide, similar to LL‑37, by fighting viruses and binding harmful amyloid‑β, but it can also be hijacked by viruses like HSV‑1. This is mostly basic science and doesn’t give any direct tips for supplementing or dosing.

Utility 2
pubmed Mar 5, 2019

Cathelicidin LL-37 restoring glucocorticoid function in smoking and lipopolysaccharide-induced airway inflammation in rats.

Weng. Jian-Zhen JZ; Wang. Yan Y; Sun. Tie-Ying TY

In rats with COPD‑like lung damage caused by cigarette smoke and bacterial toxin, giving the natural peptide LL‑37 together with a steroid inhaler (budesonide) made the steroid work better. LL‑37 boosted a protein called HDAC2 that helps steroids reduce inflammation and lowered activity of a signaling pathway (PI3K/Akt) that normally makes steroids less effective.

Utility 2
pubmed Apr 12, 2019

Cathelicidin is a "fire alarm", generating protective NLRP3-dependent airway epithelial cell inflammatory responses during infection with Pseudomonas aeruginosa.

McHugh. Brian J BJ; Wang. Rongling R; Li. Hsin-Ni HN; Beaumont. Paula E PE; Kells. Rebekah R; Steven...

The study shows that the natural peptide LL‑37 can get inside lung cells infected with the tough bacteria Pseudomonas aeruginosa and then act like a fire alarm, turning on an immune alarm system (the NLRP3 inflammasome). This triggers the release of inflammatory signals (IL‑1β, IL‑18), kills the infected cells in a controlled way, and calls in neutrophils to help clear the infection.

Utility 2
pubmed Oct 26, 2018

Citron Essential Oils Alleviate the Mediators Related to Rosacea Pathophysiology in Epidermal Keratinocytes.

Jeon. Hyeon Woo HW; Na. Eui Young EY; Yun. Sook Jung SJ; Lee. Seung-Chul SC; Lee. Jee-Bum JB

Scientists tested citron essential oil on human skin cells in the lab and found it lowered several inflammation‑related proteins that are linked to rosacea, suggesting the oil could help calm rosacea symptoms, though the study was only done in cell cultures, not people.

Utility 2
pubmed Jul 5, 2019

Antimicrobial peptides, disease severity and exacerbations in bronchiectasis.

Sibila. Oriol O; Perea. Lídia L; Cantó. Elisabet E; Shoemark. Amelia A; Cassidy. Diane D;...

In people with bronchiectasis, the natural antimicrobial peptide LL‑37 is found in higher amounts in the lungs, especially in those who get frequent flare‑ups and have worse lung function. Higher LL‑37 (and lower levels of another peptide, SLPI) predicts quicker and more frequent worsening of the disease.

Utility 2
pubmed May 5, 2018

LL-37-induced human osteoblast cytotoxicity and permeability occurs independently of cellular LL-37 uptake through clathrin-mediated endocytosis.

Anders. Emma E; Dahl. Sara S; Svensson. Daniel D; Nilsson. Bengt-Olof BO

LL-37, a natural antimicrobial peptide, can kill bone‑building cells (osteoblasts) and make their membranes leaky, and it does this even when the cells don’t take the peptide inside through the usual clathrin pathway. Blocking that uptake route cuts how much peptide gets inside but doesn’t stop the damage, meaning the harmful effect happens outside the cell.

Utility 2
pubmed Aug 17, 2018

Evaluation of LL-37 antimicrobial peptide derivatives alone and in combination with vancomycin against S. aureus.

Shurko. James F JF; Galega. Ralph S RS; Li. Chuxi C; Lee. Grace C GC

Researchers tested the natural antimicrobial peptide LL-37 and two shorter versions (LL-13 and LL-17) against tough Staph infections. When mixed with the antibiotic vancomycin, the short peptides helped kill vancomycin‑resistant strains and even made them sensitive to the drug again. They also stopped the bacteria from forming protective biofilm layers.

Utility 2
pubmed Oct 3, 2018

The activity and action mechanism of novel short selective LL-37-derived anticancer peptides against clinical isolates of Escherichia coli.

Aghazadeh. Hossein H; Memariani. Hamed H; Ranjbar. Reza R; Pooshang Bagheri. Kamran K

Researchers tested three short versions of the human peptide LL-37 and found that one called P38 kills drug‑resistant E. coli very well while not harming human cells. It works by punching holes in the bacterial membrane, and computer models show it sticks strongly to the outer layer of Gram‑negative bacteria. The study is still in the lab stage, but it points to P38 as a promising candidate for future infection treatments.

Utility 2
pubmed Oct 31, 2018

Phosphatase activity of the control of virulence sensor kinase CovS is critical for the pathogenesis of group A streptococcus.

Horstmann. Nicola N; Tran. Chau Nguyen CN; Brumlow. Chelcy C; DebRoy. Sruti S; Yao. Hui H; Nogueras...

The study shows that a part of the bacteria’s control system (CovS phosphatase) is needed for group A strep to survive attacks by the human antimicrobial peptide LL‑37. When this bacterial function is disabled, the bugs are far less able to cause skin infections, colonize the throat, or survive immune cells, and they become much more sensitive to LL‑37.

Utility 2
pubmed Mar 1, 2018

The effects of antimicrobial peptides WAM-1 and LL-37 on multidrug-resistant Acinetobacter baumannii.

Spencer. John J JJ; Pitts. Rowan E RE; Pearson. Rachel A RA; King. Lauren B LB

The study shows that two natural antimicrobial peptides, especially the marsupial peptide WAM‑1, can kill drug‑resistant Acinetobacter bacteria in lab tests and work better when combined with regular antibiotics, but this is still early‑stage research and not a ready‑to‑use treatment for everyday health hacks.

Utility 2
pubmed Feb 23, 2018

A Novel Biological Role for Peptidyl-Arginine Deiminases: Citrullination of Cathelicidin LL-37 Controls the Immunostimulatory Potential of Cell-Free DNA.

Wong. Alicia A; Bryzek. Danuta D; Dobosz. Ewelina E; Scavenius. Carsten C; Svoboda. Pavel P; Rapala-...

LL-37 is a natural peptide that helps immune cells sense stray DNA, but when it gets chemically altered by PAD enzymes (a process called citrullination), it can no longer bind DNA or trigger those immune cells, which may lower the risk of auto‑immune flare‑ups. The study shows the more parts of LL-37 that are citrullinated, the weaker its effect, and that swapping certain building blocks can protect its activity.

Utility 2
pubmed May 16, 2018

Dermatologic toxicity from novel therapy using antimicrobial peptide LL-37 in melanoma: A detailed examination of the clinicopathologic features.

Dolkar. Tsetan T; Trinidad. Celestine M CM; Nelson. Kelly C KC; Amaria. Rodabe N RN; Nagarajan. Priy...

A case report shows that injecting the immune‑boosting peptide LL‑37 into melanoma tumors can shrink the cancer but may cause serious skin problems like wart‑like bumps and blistering after about a month and a half. The skin issues went away once the injections stopped.

Utility 2
pubmed Feb 28, 2018

Tissue-specific Regulation of Innate Immune Responses by Human Cathelicidin LL-37.

Chieosilapatham. Panjit P; Ikeda. Shigaku S; Ogawa. Hideoki H; Niyonsaba. Francois F

LL-37 is a natural peptide that our bodies make in skin, gut, lungs and other surfaces to kill microbes and fine‑tune inflammation. It’s produced by skin cells, gut lining, immune cells and changes its activity depending on the tissue and the local microbes.

Utility 2
pubmed May 24, 2019

The contribution of IL-17 to the development of autoimmunity in psoriasis.

Furue. Masutaka M; Kadono. Takafumi T

The paper explains that psoriasis, a skin disease, is driven by an immune pathway involving IL-17. A natural protein called LL-37, which some people think about using for its antimicrobial benefits, can act as a trigger for this pathway in people with a certain genetic background, potentially worsening or causing psoriasis.

Utility 2
pubmed Apr 19, 2018

Astragalus polysaccharides exerts anti-infective activity by inducing human cathelicidin antimicrobial peptide LL-37 in respiratory epithelial cells.

Zhao. Lin L; Tan. Shuai S; Zhang. Hai H; Liu. Peng P; Tan. Yu-Zhu YZ; Li. Jia-Chuan JC; Jia. Da D; S...

Astragalus polysaccharides (APS) can boost the body's own antimicrobial peptide LL‑37 in airway cells, leading to stronger antibacterial effects in lab dishes, but this is only shown in cell cultures, not in people.