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LL-37

Cathelicidin, hCAP-18, FALL-39, CAP-18

An antimicrobial peptide of the cathelicidin family that provides innate immune defense by killing pathogens and modulating inflammation and wound healing.

Quick Stats
Studies 2230
Trials 95
Formula C205H340N60O53
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Utility 2
pubmed Feb 24, 2017

The relationship of serum vitamins A, D, E and LL-37 levels with allergic status, tonsillar virus detection and immune response.

Elenius. Varpu V; Palomares. Oscar O; Waris. Matti M; Turunen. Riitta R; Puhakka. Tuomo T; Rück...

The study found that higher vitamin D and E levels are linked to fewer allergies, while vitamin A may help fight viruses. The antimicrobial peptide LL‑37 was tied to lower levels of certain immune cells that drive inflammation. However, the research doesn’t give clear ways to boost LL‑37 or specific dosing advice for everyday use.

Utility 2
pubmed Mar 1, 2017

Mirolysin, a LysargiNase from Tannerella forsythia, proteolytically inactivates the human cathelicidin, LL-37.

Koneru. Lahari L; Ksiazek. Miroslaw M; Waligorska. Irena I; Straczek. Anna A; Lukasik. Magdalena M;...

The study shows that a gum‑disease bacterium makes an enzyme called mirolysin that can cut up the human immune peptide LL‑37, stopping it from killing bacteria and binding harmful toxins. This means that bad oral bacteria can weaken one of our natural defenses.

Utility 2
pubmed Jul 15, 2016

Antimicrobial peptide LL-37 promotes the proliferation and invasion of skin squamous cell carcinoma by upregulating DNA-binding protein A.

Wang. Wei W; Jia. Jinjing J; Li. Changji C; Duan. Qiqi Q; Yang. Jiao J; Wang. Xin X; Li. Ruilian R;...

The study shows that the antimicrobial peptide LL-37 can actually help skin squamous cell cancer cells grow and spread by turning on a protein called dbpA through the NF‑κB pathway, and blocking this pathway or the protein reduces the cancer‑like behavior.

Utility 2
pubmed Jul 30, 2016

Pathogenesis of antimicrobial peptides LL-37 and CpG-ODN in ANCA associated vasculitis.

Luan. Jun-Jun JJ; Xing. Guang-Qun GQ

The study found that people with a certain autoimmune disease (AAV) have higher levels of the antimicrobial peptide LL‑37 and the immune signal IFN‑α, and that exposing immune cells to LL‑37 (especially together with DNA fragments called CpG‑ODN) makes them release even more IFN‑α and produce auto‑antibodies. This suggests LL‑37 can boost inflammation in people prone to autoimmunity.

Utility 2
pubmed Jul 4, 2017

Formulation and candidacidal activity of magnetic nanoparticles coated with cathelicidin LL-37 and ceragenin CSA-13.

Niemirowicz. Katarzyna K; Durnaś. Bonita B; Tokajuk. Grażyna G; Piktel. Ewelina E; Micha...

Scientists attached the natural antimicrobial peptide LL‑37 (and a synthetic version called CSA‑13) to tiny magnetic particles. This combo killed several Candida yeast strains even better than the free peptides, worked in body‑like fluids, stopped biofilm formation, and didn’t hurt bone‑cell growth, showing it’s safe in the lab.

Utility 2
pubmed Nov 5, 2017

Functionalized PVA-silk blended nanofibrous mats promote diabetic wound healing via regulation of extracellular matrix and tissue remodelling.

Chouhan. Dimple D; Janani. G G; Chakraborty. Bijayashree B; Nandi. Samit K SK; Mandal. Biman B BB

Scientists made a thin, stretchy dressing that slowly releases the antimicrobial peptide LL‑37 and a special silk protein from a non‑mulberry silkworm. In diabetic rabbits, this dressing helped wounds close faster, grew new blood vessels and skin more quickly, and made the healed skin stronger than standard dressings.

Utility 2
pubmed Sep 23, 2016

Pyrazole derived ultra-short antimicrobial peptidomimetics with potent anti-biofilm activity.

Ahn. Mija M; Gunasekaran. Pethaiah P; Rajasekaran. Ganesan G; Kim. Eun Young EY; Lee. Soo-Jae SJ; Ba...

Researchers made a tiny synthetic peptide called Py11 that kills tough bacteria better than the natural peptide LL‑37 and the bee‑sting peptide melittin. It’s more stable in the body, works even in salty conditions, breaks down bacterial membranes, stops biofilm formation, and reduces inflammation caused by bacterial toxins. However, it’s still a lab‑only compound and not something you can buy or safely use yet.

Utility 2
pubmed May 12, 2017

Circulating cathelicidin levels correlate with mucosal disease activity in ulcerative colitis, risk of intestinal stricture in Crohn's disease, and clinical prognosis in inflammatory bowel disease.

Tran. Diana Hoang-Ngoc DH; Wang. Jiani J; Ha. Christina C; Ho. Wendy W; Mattai. S Anjani SA; Oikonom...

A study found that the blood level of the natural peptide LL‑37 goes down when ulcerative colitis or Crohn's disease gets worse, and higher levels are linked to better recovery and lower risk of intestinal narrowing. Checking LL‑37 together with the usual inflammation marker CRP gives a clearer picture of gut inflammation than CRP alone.

Utility 2
pubmed Nov 1, 2017

The cathelicidin protein CRAMP is a potential atherosclerosis self-antigen in ApoE(-/-) mice.

Mihailovic. Peter M PM; Lio. Wai Man WM; Yano. Juliana J; Zhao. Xiaoning X; Zhou. Jianchang J; Chyu....

In mice that are prone to artery plaque buildup, a short piece of the natural antimicrobial protein CRAMP (the mouse version of human LL‑37) can act like a self‑antigen. Giving a low dose of this peptide reduced plaque formation, while a high dose made plaques worse and attracted more immune cells.

Utility 2
pubmed Jun 27, 2017

Stiffening of bacteria cells as a first manifestation of bactericidal attack.

Pogoda. Katarzyna K; Piktel. Ewelina E; Deptuła. Piotr P; Savage. Paul B PB; Lekka. Małg...

The study shows that the human antimicrobial peptide LL-37 (and similar synthetic compounds) makes bacterial cells temporarily harder (stiffer) before they become softer, and this change depends on how much of the compound is used and how long it’s applied. Measuring how stiff bacteria get could be a new way to track how well these membrane‑targeting antimicrobials work.

Utility 2
pubmed Nov 6, 2017

Membrane interactions of microgels as carriers of antimicrobial peptides.

Nordström. Randi R; Nyström. Lina L; Andrén. Oliver C J OCJ; Malkoch. Michael M; Umer...

The study shows that negatively charged microgel particles can hold a lot of the antimicrobial peptide LL‑37 and keep it safe from enzymes that would break it down. However, the microgels themselves don’t kill bacteria – the peptide has to be released first. Releasing the peptide faster (by using a shorter version or a less strongly charged microgel) makes the antibacterial effect stronger, and the formulation appears safe for red blood cells.

Utility 2
pubmed Mar 1, 2017

Linear antimicrobial peptides with activity against herpes simplex virus 1 and Aichi virus.

Vilas Boas. Liana Costa Pereira LC; de Lima. Lídia Maria Pinto LM; Migliolo. Ludovico L; Mendes...

The study tested 10 lab-made antimicrobial peptides and found that two of them, LL‑37 (a human peptide) and Pa‑MAP (from a fish), can block viruses in cell experiments – LL‑37 stopped Aichi virus by about 96% and Pa‑MAP stopped herpes simplex virus‑1 by about 90%. The other peptides didn’t work well.

Utility 2
pubmed Oct 27, 2017

Immunoprecipitation high performance liquid chromatographic analysis of healing process in chronic suppurative osteomyelitis of the jaw.

Kim. Soung Min SM; Eo. Mi Young MY; Cho. Yun Ju YJ; Kim. Yeon Sook YS; Lee. Suk Keun SK

In patients with a serious jaw bone infection, researchers tracked healing by measuring proteins in wound fluid after surgery. They saw a small rise in the antimicrobial peptide LL‑37 along with other immune signals, while inflammation markers linked to bacteria went down. By day two, proteins that help build new bone and tissue were higher, suggesting the wound was healing well.

Utility 2
pubmed Oct 31, 2017

Role of Soluble Innate Effector Molecules in Pulmonary Defense against Fungal Pathogens.

Ordonez. Soledad R SR; Veldhuizen. Edwin J A EJA; van Eijk. Martin M; Haagsman. Henk P HP

LL-37 is a natural peptide in our lungs that can kill fungi in lab tests, but the real lung environment – with its salt levels, acidity, and mucus – can weaken this effect. Other lung proteins also help fight fungi, but we still don’t know how they work together with LL-37 in the body.

Utility 2
pubmed Jan 9, 2017

The dual role of cathelicidins in systemic inflammation.

Pinheiro da Silva. Fabiano F; Machado. Marcel Cerqueira César MC

LL-37 is a natural peptide that kills microbes and also signals the immune system, but it can either increase or decrease inflammation depending on the situation. Lab studies show that giving extra LL-37 from outside doesn’t always act like the body’s own version, and its effects change with the disease environment.

Utility 2
pubmed Nov 20, 2017

RNase 7 Strongly Promotes TLR9-Mediated DNA Sensing by Human Plasmacytoid Dendritic Cells.

Kopfnagel. Verena V; Wagenknecht. Sylvia S; Harder. Jürgen J; Hofmann. Karsten K; Kleine. Micha...

The study shows that RNase 7, a natural skin protein, helps immune cells in the skin spot DNA from our own cells and from bacteria much faster, leading to a strong antiviral signal that can block herpes virus infection. It works better than other similar skin peptides like LL‑37.

Utility 2
pubmed Aug 16, 2016

Cinobufagin Modulates Human Innate Immune Responses and Triggers Antibacterial Activity.

Xie. Shanshan S; Spelmink. Laura L; Codemo. Mario M; Subramanian. Karthik K; Pütsep. Katrin K;...

The study shows that cinobufagin, a compound from the traditional medicine Chan‑Su, can dampen certain immune cell activations while also boosting the release of natural antibiotics like LL‑37 from neutrophils, making immune cells better at killing bacteria. However, cinobufagin is a potent toxin and not proven safe for self‑administration, so the findings are more about understanding mechanisms than giving a ready‑to‑use supplement protocol.

Utility 2
pubmed 2016

The Roles of Cathelicidin LL-37 in Inflammatory Bowel Disease.

Sun. Lihua L; Wang. Wensheng W; Xiao. Weidong W; Yang. Hua H

LL-37 is a natural peptide made by immune cells and gut lining that helps fight infections and supports healing. In ulcerative colitis, its levels are higher in both sick and healthy tissue, while in Crohn's disease its levels don't change. Animal and early human studies suggest LL-37 can protect the gut, hinting it might become a future treatment for inflammatory bowel disease.