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Kisspeptin-10

KP-10, Metastin (45-54), Kisspeptin-10 (human), KiSS-1

Kisspeptin-10 is a decapeptide that activates the KISS1R receptor to stimulate GnRH release, regulating the reproductive hormone axis and fertility.

Quick Stats
Studies 877
Trials 47
Formula C63H83N17O14
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Utility 2
pubmed Apr 17, 2019

Kisspeptin and body weight homeostasis in relation to phenotypic features of polycystic ovary syndrome; metabolic regulation of reproduction.

Rashad. Nearmeen M NM; Al-Sayed. Radwa M RM; Yousef. Mohammed S MS; Saraya. Yasser S YS

The study found that women with polycystic ovary syndrome (PCOS) have higher blood levels of the hormone kisspeptin than women without PCOS, but within the PCOS group, kisspeptin levels drop as body weight and BMI go up. Lower kisspeptin was linked to worse blood sugar, cholesterol, and body‑fat measures, while higher levels were tied to certain PCOS traits like hirsutism and hormone levels.

Utility 2
pubmed May 2, 2019

Circulatory metastin/kisspeptin-1 in polycystic ovary syndrome: a systematic review and meta-analysis with diagnostic test accuracy.

Varikasuvu. Seshadri Reddy SR; Prasad. V Satya VS; Vamshika. V C VC; Satyanarayana. M V MV; Panga. J...

People with polycystic ovary syndrome (PCOS) tend to have higher levels of the hormone kisspeptin-1 in their blood. The study found that kisspeptin-1 levels are linked to other PCOS‑related markers like AMH, testosterone, and DHEA, and that measuring kisspeptin‑1 could help identify PCOS with fairly good accuracy.

Utility 2
pubmed Aug 19, 2016

An update on the prevention of ovarian hyperstimulation syndrome.

El Tokhy. Omar O; Kopeika. Julia J; El-Toukhy. Tarek T

The paper reviews new ways to stop a dangerous side‑effect of IVF called ovarian hyperstimulation syndrome (OHSS). It highlights that using kisspeptin or a GnRH‑agonist instead of the usual hCG hormone to finish egg maturation, lowering the hCG dose, freezing all embryos and transferring them later, or giving dopamine‑type drugs after egg collection can all cut the risk of OHSS.

Utility 2
pubmed Jan 31, 2017

Effect of supplements during the cold season on the reproductive system in prepubertal Tibetan sheep ewes.

Jing. Xiaoping X; Peng. Quanhui Q; Hu. Rui R; Wang. Hongze H; Yu. Xiaoqiang X; Degen. Allan A; Zou....

Feeding pre‑pubertal Tibetan sheep extra nutrients during the cold season (either concentrate feed or mineral lick blocks) helped them grow faster, increased the number of ovarian follicles, and raised key reproductive hormones. The study also showed that better nutrition turned up the activity of the kisspeptin (KiSS‑1) system in the brain, which in turn boosted the whole reproductive hormone cascade.

Utility 2
pubmed May 4, 2017

Investigational and experimental GnRH analogs and associated neurotransmitters.

Blumenfeld. Zeev Z

This review talks about drugs that mimic or block the hormone GnRH, which controls sex hormone production. It lists many medical reasons these drugs are used, like prostate or breast cancer, fertility issues, and hormone‑related conditions. It also mentions newer compounds like kisspeptin‑10 and neurokinin B that act on the same system and could be fine‑tuned for better results with fewer side effects.

Utility 2
pubmed Apr 13, 2015

Rational design of triazololipopeptides analogs of kisspeptin inducing a long-lasting increase of gonadotropins.

Beltramo. Massimiliano M; Robert. Vincent V; Galibert. Mathieu M; Madinier. Jean-Baptiste JB; Marcea...

Scientists made new kisspeptin‑10‑based molecules that resist breakdown and stay in the blood longer. In sheep, the best of these compounds caused a much longer rise in the hormones that trigger ovulation compared to the natural peptide. This shows the design works, but it’s still an early animal study and not ready for human use.

Utility 2
pubmed Dec 12, 2016

Dynamic Regulation of Hypothalamic DMXL2, KISS1, and RFRP Expression During Postnatal Development in Non-Human Primates.

Wahab. Fazal F; Drummer. Charis C; Schlatt. Stefan S; Behr. Rüdiger R

The study looked at how the brain chemicals kisspeptin, DMXL2, and a hormone‑blocking factor (RFRP) change as baby marmoset monkeys grow up. It found that kisspeptin and DMXL2 levels are low at birth, rise during early childhood, dip before puberty, then go up again at puberty and adulthood. The hormone‑blocking factor spikes just before puberty. This tells us that kisspeptin naturally increases when the reproductive system is maturing, but the research was done in monkeys and didn’t test any treatments in people.

Utility 2
pubmed Jul 16, 2015

Uterine and placental KISS1 regulate pregnancy: what we know and the challenges that lie ahead.

Babwah. Andy V AV

This review explains that a protein called kisspeptin, made by the uterus and placenta, helps early pregnancy steps like embryo implantation, and that messing with its signals can cause infertility. Most of the evidence comes from mice, and the findings don’t always match what happens in humans, so we can’t directly apply it yet.

Utility 2
pubmed Jun 18, 2015

Direct comparison of the effects of intravenous kisspeptin-10, kisspeptin-54 and GnRH on gonadotrophin secretion in healthy men.

Jayasena. C N CN; Abbara. A A; Narayanaswamy. S S; Comninos. A N AN; Ratnasabapathy. R R; Bassett. P...

In a small study of healthy men, giving kisspeptin‑10 or kisspeptin‑54 through an IV raised the hormones LH and FSH, but not as strongly as the standard hormone GnRH. Both kisspeptin forms worked about the same, and the effect grew with higher doses.

Utility 2
pubmed Dec 1, 2015

Kisspeptin signalling and its roles in humans.

Tng. Eng Loon EL

Kisspeptin-10 is a small protein that helps control the brain's reproductive hormone system. It works with estrogen and leptin to trigger puberty and fertility, and scientists think it could be used for things like fertility testing, birth control, and treating hormone‑driven cancers, but there are no clear dosing or DIY protocols yet.

Utility 2
pubmed Sep 27, 2016

Expression of GnRH and Kisspeptin in Primary Cultures of Fetal Rat Brain.

Sukhbaatar. Unurjargal U; Kanasaki. Haruhiko H; Mijiddorj. Tselmeg T; Oride. Aki A; Hara. Tomomi T;...

In a lab dish made from fetal rat brain cells, the short peptide kisspeptin‑10 was shown to boost the production of the hormone‑messenger GnRH, and both kisspeptin and GnRH can increase their own levels. Estrogen (E2) raised kisspeptin but not GnRH. This confirms that kisspeptin can directly stimulate GnRH‑producing neurons, at least in this early‑stage animal model.

Utility 2
pubmed 2015

[Kisspeptin and leptin in the regulation of fertility].

Pankov. Yu A YA

The study shows that kisspeptin and its receptor are key players in starting puberty and controlling fertility, and that certain gene mutations can cause early or delayed puberty. It also finds that the hormone leptin, which reflects body fat levels, can turn on kisspeptin in the brain, helping trigger the reproductive hormone cascade. For biohackers, this means body‑fat‑related leptin levels can influence reproductive health, but the paper doesn’t give any direct ways to use kisspeptin as a supplement or protocol.

Utility 2
pubmed Sep 28, 2016

Plasma kisspeptin and ghrelin levels are independently correlated with physical activity in patients with anorexia nervosa.

Hofmann. Tobias T; Elbelt. Ulf U; Haas. Verena V; Ahnis. Anne A; Klapp. Burghard F BF; Rose. Matthia...

In a small study of women with anorexia nervosa, higher blood levels of the hormone kisspeptin were linked to fewer daily steps, while higher ghrelin levels were linked to more steps. Other hormones studied didn’t show a clear connection to activity.

Utility 2
pubmed Sep 16, 2016

Interactions Between Neurokinin B and Kisspeptin in Mediating Estrogen Feedback in Healthy Women.

Skorupskaite. Karolina K; George. Jyothis T JT; Veldhuis. Johannes D JD; Millar. Robert P RP; Anders...

In healthy women, blocking the neurokinin‑3 receptor (NK3R) lowers the natural pulse of the hormone LH, while a short infusion of the peptide kisspeptin‑10 can still boost LH even when estrogen is trying to suppress it. The LH rise lasts beyond the infusion, and the interaction shows that neurokinin B works upstream of kisspeptin in controlling estrogen feedback.

Utility 2
pubmed 2014

Kisspeptin-10 induces endothelial cellular senescence and impaired endothelial cell growth.

Usui. Sayaka S; Iso. Yoshitaka Y; Sasai. Masahiro M; Mizukami. Takuya T; Mori. Hiroyoshi H; Watanabe...

Kisspeptin‑10, a peptide that’s being studied for various roles, was found to slow down blood‑vessel growth and make endothelial cells act old and stop dividing. In rats with blocked blood flow, it reduced recovery, and in human vein cells it blocked tube formation and caused senescence markers to rise. A drug that blocks Rho‑kinase could partly reverse these bad effects.

Utility 2
pubmed Nov 17, 2014

Differential stimulation of insulin secretion by GLP-1 and Kisspeptin-10.

Schwetz. Tara A TA; Reissaus. Christopher A CA; Piston. David W DW

The study shows that kisspeptin‑10 (KP) can boost insulin release from pancreatic beta cells in mice, but it does so by changing the cells' metabolism rather than by the usual calcium signal that most insulin‑boosting drugs use. GLP‑1 works through a different pathway that involves a G‑protein alpha subunit. These two peptides act via distinct mechanisms, and blocking the G‑protein beta‑gamma part stops KP’s effect but not GLP‑1’s.

Utility 2
pubmed Feb 14, 2015

Associations of cadmium, bisphenol A and polychlorinated biphenyl co-exposure in utero with placental gene expression and neonatal outcomes.

Xu. Xijin X; Chiung. Yin Mei YM; Lu. Fangfang F; Qiu. Shaoshan S; Ji. Minhui M; Huo. Xia X

The study found that pregnant women exposed to cadmium, BPA, and PCBs had higher levels of the KISS1 gene (which makes the kisspeptin peptide) in their placentas, and this was linked to lower birth weight. The toxic chemicals also raised leptin signals, but only BPA was tied to those changes. In short, environmental pollutants can boost kisspeptin production in the womb, which may affect growth.

Utility 2
pubmed Apr 13, 2014

The Warburg effect in tumor progression: mitochondrial oxidative metabolism as an anti-metastasis mechanism.

Lu. Jianrong J; Tan. Ming M; Cai. Qingsong Q

The paper explains that cancer cells often avoid using their mitochondria for energy (the Warburg effect) so they produce less harmful reactive oxygen species (ROS) and become more resistant to a type of cell death that stops metastasis. Forcing cancer cells to burn glucose in the mitochondria raises ROS, making them vulnerable again. The peptide KISS1 (kisspeptin) is mentioned as a natural factor that can push cells toward mitochondrial metabolism, potentially suppressing spread of tumors.

Utility 2
pubmed Mar 4, 2013

LC-MS/MS quantification of a neuropeptide fragment kisspeptin-10 (NSC 741805) and characterization of its decomposition product and pharmacokinetics in rats.

Liu. Zhongfa Z; Ren. Chen C; Jones. William W; Chen. Ping P; Seminara. Stephanie B SB; Chan. Yee-Min...

The study shows that kisspeptin-10, a short hormone fragment that can boost reproductive hormones, breaks down very quickly in the blood of rats—only a few minutes at room temperature and disappears within half an hour after an injection. This means it’s hard to keep active long enough to have a lasting effect without special delivery methods.

Utility 2
pubmed Jan 4, 2015

KISS1 can be used as a novel target for developing a DNA immunocastration vaccine in ram lambs.

Han. Yanguo Y; Liu. Guiqiong G; Jiang. Xunping X; Ijaz. Nabeel N; Tesema. Birhanu B; Xie. Guangyue G

Scientists made a DNA vaccine that teaches a sheep's immune system to attack the KISS1 protein, which normally helps start puberty and keep fertility normal. The vaccinated lambs made strong anti‑KISS1 antibodies, had lower testosterone, smaller testes, and showed less sexual behavior compared to untreated lambs.