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Kisspeptin-10

KP-10, Metastin (45-54), Kisspeptin-10 (human), KiSS-1

Kisspeptin-10 is a decapeptide that activates the KISS1R receptor to stimulate GnRH release, regulating the reproductive hormone axis and fertility.

Quick Stats
Studies 877
Trials 47
Formula C63H83N17O14
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Utility 1
pubmed Apr 21, 2016

Placental Kisspeptins Differentially Modulate Vital Parameters of Estrogen Receptor-Positive and -Negative Breast Cancer Cells.

Rasoulzadeh. Zahra Z; Ghods. Roya R; Kazemi. Tohid T; Mirzadegan. Ebrahim E; Ghaffari-Tabrizi-Wizsy....

Researchers found that kisspeptin-10 from the placenta can change how breast cancer cells grow and move in lab dishes, but the effects differ between estrogen‑positive and estrogen‑negative cells. The peptide slowed cell growth, altered stickiness, and changed inflammation signals, yet it also made some cancer cells more invasive. These results are from cell‑culture experiments, not human studies, so they don’t translate into any direct health or anti‑cancer protocol for everyday use.

Utility 1
pubmed Mar 2, 2017

Evaluation of the correlation of vasculogenic mimicry, ALDH1, KiSS-1, and MACC1 in the prediction of metastasis and prognosis in ovarian carcinoma.

Yu. Lan L; Zhu. Bo B; Wu. Shiwu S; Zhou. Lei L; Song. Wenqing W; Gong. Xiaomeng X; Wang. Danna D

The study looked at how levels of a protein called KiSS-1 (related to the peptide kisspeptin) and other markers are linked to ovarian cancer spread and patient survival. Higher KiSS-1 levels were tied to better outcomes, while lower levels were linked to worse prognosis.

Utility 1
pubmed 2016

Evaluation of KiSS1 as a Prognostic Biomarker in North Indian Breast Cancer Cases.

Singh. Richa R; Bhatt. Madan Lal Brahma ML; Singh. Saurabh Pratap SP; Kumar. Vijay V; Goel. Madhu Ma...

The study looked at the KiSS1 gene (which makes the peptide kisspeptin) in breast cancer tissue and found that higher levels are seen in tumors compared to normal tissue, but lower levels are linked to metastasis and worse survival.

Utility 1
pubmed 2016

Expression of preoperative KISS1 gene in tumor tissue with epithelial ovarian cancer and its prognostic value.

Cao. Fang F; Chen. Liping L; Liu. Manhua M; Lin. Weiwei W; Ji. Jinlong J; You. Jun J; Qiao. Fenghai...

The study found that higher levels of the kisspeptin gene (KISS1) in ovarian cancer tumors are linked to better survival, meaning the gene might be a useful marker to predict outcomes, but it doesn’t tell you how to use kisspeptin as a supplement or therapy.

Utility 1
pubmed Nov 24, 2014

Kisspeptin-10 inhibits the migration of breast cancer cells by regulating epithelial-mesenchymal transition.

Song. Guo-Qing GQ; Zhao. Yi Y

Researchers found that a short piece of the protein kisspeptin, called KP-10, can kill breast cancer cells and stop them from spreading in lab dishes and in mice, mainly by blocking a process called EMT that helps cancer cells move. However, the work is early‑stage, done in cell cultures and animal models, and doesn’t give any guidance on safe human use.

Utility 1
pubmed Feb 23, 2016

Relationships between leptin, KiSS-1/GPR54 expression and TSH secretion from pituitary cells of pubertal ewes in vitro.

Radwańska. Paulina P; Kosior-Korzecka. Urszula U

In a lab study using cells from young sheep, researchers found that the hormone leptin can directly boost the release of kisspeptin-10 and increase the expression of its receptor (GPR54) in pituitary cells. However, adding kisspeptin-10 itself did not meaningfully change thyroid‑stimulating hormone (TSH) levels, except for a brief effect after two hours.

Utility 1
pubmed Aug 15, 2014

Kisspeptin-10 potentiates miniature excitatory postsynaptic currents in the rat supraoptic nucleus.

Yokoyama. Toru T; Minami. Kouichiro K; Terawaki. Kiyoshi K; Miyano. Kanako K; Ogata. Junichi J; Maru...

In rats, a short form of the hormone‑related peptide kisspeptin‑10 makes certain brain cells that control water balance fire more often, but it doesn’t change how strong each signal is. This effect depends on the kisspeptin receptor and a signaling pathway called PKC.

Utility 1
pubmed Aug 21, 2013

The Kiss-1/Kiss-1R complex as a negative regulator of cell motility and cancer metastasis (Review).

Ji. Ke K; Ye. Lin L; Mason. Malcolm D MD; Jiang. Wen G WG

This review explains that the protein made by the Kiss-1 gene and its related peptides (like kisspeptin‑10) can slow down the spread of cancer cells by reducing their ability to move and invade other tissues, but it doesn’t give any practical advice on how to use these peptides for health or performance purposes.

Utility 1
pubmed Jun 26, 2017

In vitro and in vivo effects of kisspeptin antagonists p234, p271, p354, and p356 on GPR54 activation.

Albers-Wolthers. C H J CHJ; de Gier. J J; Walen. M M; van Kooten. P J S PJS; Lambalk. C B CB; Leegwa...

The study tested four kisspeptin‑10 antagonist peptides (p234, p271, p354, p356) in cells and in female dogs and found that none of them blocked kisspeptin signaling or reduced luteinizing hormone (LH) levels. In other words, these compounds do not work as kisspeptin blockers in the tested models.

Utility 1
pubmed Apr 20, 2016

Circulating levels of leptin, nesfatin-1 and kisspeptin in postmenopausal obese women.

Çelik. Feyza F; Belviranli. Muaz M; Okudan. Nilsel N

In a study of 83 women, researchers found that while obese women had higher leptin levels, the amounts of the hormones nesfatin-1 and kisspeptin (including kisspeptin-10) in the blood were the same regardless of whether the women were obese or not, and whether they were pre‑ or post‑menopausal.

Utility 1
pubmed Feb 2, 2016

Overexpression of KiSS-1 reduces colorectal cancer cell invasion by downregulating MMP-9 via blocking PI3K/Akt/NF-κB signal pathway.

Chen. Shaoqin S; Chen. Wei W; Zhang. Xiang X; Lin. Suyong S; Chen. Zhihua Z

Scientists forced colorectal cancer cells to make more of a protein called KiSS-1 and found that these cells grew slower, were less able to invade, and died more often. This effect happened because KiSS-1 lowered the levels of a molecule (MMP-9) that helps cancer spread, by shutting down a signaling chain called PI3K/Akt/NF‑κB. When they added drugs that turn that signaling chain back on, the anti‑cancer effect of KiSS-1 went away.

Utility 1
pubmed May 11, 2017

Plasma kisspeptin levels in lactational amenorrhea.

Kotani. Masato M; Katagiri. Fumihiko F; Hirai. Tsuyoshi T; Kagawa. Jiro J; Tanaka. Issei I

The study measured the amount of the hormone‑like peptide kisspeptin in the blood of women who were not having periods because they were breastfeeding (lactational amenorrhea) and compared it to women with normal cycles and a few women with other types of amenorrhea. Kisspeptin levels were about the same in breastfeeding women as in normal‑cycle women, and varied in the pathological cases, suggesting that circulating kisspeptin isn’t dramatically lowered during lactational or some pathological amenorrhea.

Utility 1
pubmed Oct 7, 2016

Defining Subpopulations of Arcuate Nucleus GABA Neurons in Male, Female, and Prenatally Androgenized Female Mice.

Marshall. Christopher J CJ; Desroziers. Elodie E; McLennan. Timothy T; Campbell. Rebecca E RE

The study looked at which chemicals are found together with GABA‑producing neurons in a brain area that controls things like hunger and reproduction. It found that kisspeptin, the peptide you asked about, is almost never present in these GABA neurons, while other chemicals like NPY are much more common.

Utility 1
pubmed Feb 11, 2014

Coevolution of the spexin/galanin/kisspeptin family: Spexin activates galanin receptor type II and III.

Kim. Dong-Kyu DK; Yun. Seongsik S; Son. Gi Hoon GH; Hwang. Jong-Ik JI; Park. Cho Rong CR; Kim. Jae I...

Scientists found that a peptide called spexin, which is related to kisspeptin and galanin, likely evolved together with those families and can turn on certain galanin receptors (GALR2/3) in humans and other animals. This is mostly a basic science discovery and doesn’t give any direct tips for health or performance.

Utility 1
pubmed Oct 30, 2014

Stimulation of δ subunit-containing GABAA receptor by DS1 increases GnRH receptor expression but reduces GnRH mRNA expression in GnRH-producing GT1-7 cells.

Sukhbaatar. Unurjargal U; Mijiddorj. Tselmeg T; Oride. Aki A; Kanasaki. Haruhiko H

The study looked at how a GABAA‑receptor drug (DS1) and the peptide kisspeptin‑10 affect hormone‑producing brain cells in a mouse cell line. DS1 boosted the cells' GnRH‑receptor levels but actually lowered the amount of GnRH hormone mRNA they made. Kisspeptin‑10 also activated some signaling pathways, and together with DS1 it gave a bigger effect on those pathways, but the work was done only in cells, not people.

Utility 1
pubmed Jul 2, 2015

Does salt have a permissive role in the induction of puberty?

Pitynski. Dori D; Flynn. Francis W FW; Skinner. Donal C DC

The abstract suggests that eating a lot of salt might make kids hit puberty earlier by messing with hormones that control reproduction, but it’s just a theory – no experiments were done yet.

Utility 1
pubmed 2013

Kisspeptin and puberty in mammals.

Terasawa. Ei E; Guerriero. Kathryn A KA; Plant. Tony M TM

The paper explains that kisspeptin cells in a brain area called the arcuate nucleus are essential for starting puberty because they help generate the hormone pulses that trigger reproductive development, but they probably don’t set the timing of puberty themselves.

Utility 1
pubmed May 27, 2014

Insulin: its role in the central control of reproduction.

Sliwowska. Joanna H JH; Fergani. Chrysanthi C; Gawałek. Monika M; Skowronska. Bogda B; Fichna....

This review explains that insulin, a hormone we usually think of for blood sugar control, also talks to the brain areas that control reproduction. It looks at animal studies, how insulin exposure before birth can affect adult fertility, and some human data linking insulin problems to reproductive issues. However, it doesn’t give any specific tips or dosages for using insulin or related peptides like kisspeptin‑10 in everyday health hacks.